Atlas of Cardiac Innervation by Vasken Dilsizian, Jagat Narula

By Vasken Dilsizian, Jagat Narula

This atlas elucidates the position of the neuronal part in common cardiovascular functionality and cardiac problems and discusses the presently on hand imaging objectives and probes. It presents a origin in cardiac neuronal imaging and photograph processing and serves as a advisor within the powerful usage of those suggestions in scientific and learn settings. The atlas discusses the rules of autonomic keep watch over within the legislation of cardiac functionality and affliction; chemistry and biology of radiotracers designed to focus on adjustments within the myocardial sympathetic and parasympathetic innervation as a functionality of affliction or therapy; neuronal imaging in center failure and opposite home improvement and using puppy imaging in quantification of cardiac innervation; cardiac sympathetic innervation in ventricular arrhythmias and equipment remedy; stipulations that have an effect on the autonomic fearful process; and the function of myocardial blood circulation and cardiac neuronal imaging in denervation and reinnervation in cardiac transplant recipients. that includes full-color illustrations, schematic diagrams, and diagnostic algorithms, Atlas of Cardiac Innervation is a priceless source for cardiologists, radiologists, nuclear medication physicians, and electrophysiologists.

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Scale bar: 50 μm. (b) Quantification of mean stellate ganglia neuronal size from normal, ICM, and NICM hearts. In addition to neuronal enlargement, synaptic density was significantly increased in stellate ganglia from NICM compared with normal hearts [49]. These findings may be a consequence of reflex sympathoexcitation resulting from myocardial injury (Adapted from Ajijola et al. [49]). b ms BSG Stimulation Figure 2-16. Functional remodeling of sympathetic innervation after myocardial infarction.

In another study in a canine pacinginduced HF model, VNS-treated dogs had improved ANS control of cardiac function, indicated by increased heart rate variability, baroreflex sensitivity, and reduced plasma norepinephrine levels [76]. Plasma angiotensin II and C-reactive protein levels also were decreased in the Figure 2-17. (continued) treatment group, suggesting that VNS may inhibit the renin–angiotensin–aldosterone system and exert an anti-inflammatory effect, respectively [76]. Based on the promising results of these preclinical studies, VNS has become the focus of three major HF clinical trials: CardioFit [77], Neural Cardiac Therapy for Heart Failure Study (NECTAR-HF) [78], and Autonomic Neural Regulation Therapy to Enhance Myocardial Function in Heart Failure (Anthem-HF) [9].

Int J Cardiol. 2013;168:86–93. 73.  Vagal nerve stimulation: a review of its applications and potential mechanisms that mediate its clinical effects. Neurosci Biobehav Rev. 2005;29:493–500. 74. Ikramuddin S, Blackstone RP, Brancatisano A, Toouli J, Shah SN, Wolfe BM, Fujioka K, Maher JW, Swain J, Que FG, Morton JM, Leslie DB, Brancatisano R, Kow L, O’Rourke RW, Deveney C, 85.  Neural regulation of renin release: experimental Takata M, Miller CJ, Knudson MB, Tweden KS, Shikora SA, Sarr evidence and clinical implications in arterial hypertension.

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